Re: Critique of ID & No Free Lunch

From: Josh Bembenek (jbembe@hotmail.com)
Date: Tue Nov 12 2002 - 12:31:47 EST

  • Next message: Darryl Maddox: "Re: Religious Right in Texas"

    Dr. Campbell-

    "As a rule, this method will tell us whether the random probability is so
    low as to make another explanation preferable, rather than being able to
    truly rule out random events. The only exception is if we can truly assign
    a zero probability to something."

    If we take this reasoning, we should accept nicely Dawkins explanation of
    miracle-like events in "The Blind Watchmaker," where even if we saw a stone
    statue waving its hand at us, we should not suspect miracle. Rather we
    should conclude that an extremely low probability event occured where the
    random thermal movement of all atoms in the arm of the statue move
    simultaneously in a given direction to produce the effect. Dembski's method
    leads us to the best inference, given that we allow supernatural
    explanations to be considered among the possibilities, and I think it is a
    valuable tool.

    "The complexity must be pre-specified, an issue that Dembski has noted but
    not solved to my satisfaction.
    hwkurgtrawhgtuszhrgturehbgjsdfhgbhfgbrbrewbfwbfrshgbrer is a complex
    sequence of letters, and could be made more complex by my randomly banging
    on the keys for longer. The probability that I would generate that exact
    sequence of letters is very low. However, the probability that I would
    generate some sequence of letters was 1, assuming that the computer works.
    Only by having a narrow goal, not defined with reference to the results, can
    the random interpretation be adequately tested. Without a good idea of the
    relevant probabilities, we cannot tell if we have a decent test case, much
    less the result of the test."

    This reflects very closely to a discussion we were having previously, which
    was abandoned based upon the difference of how we interpret protein
    function. It also brings up a line of thinking that I need to do some
    investigations into, as someone on the list-serve graciously sent me some
    primary-literature references concerning this exact topic. Regardless, I
    think the essence of this problem is: Just how specified is biological
    activity and function among protein sequences? The critical distinction
    between the analogy of your computer sequence of letters and the formation
    of life and the generation of biological complexity and information is the
    fact that although a computer can generate any sequence of letters, and this
    can be considered specified in any kind of way, biological sequences must be
    specified in a biologically relevant way: sequences must confer functional
    advantages that lead to evolutionarily selective adaptations. So the
    ultimate and crucial question is exactly how narrow is this goal? My
    opinion, not greatly supported by exhaustive research, but rather my
    inclinations developed from structural biology and biochemistry states that
    this goal is in fact quite narrow and that we can expect specified
    complexity as assigned to biological function to be quite narrow.

    This is the essence of the problem as applied to biological information:

    Consider three inter-related "landscapes" of all possible identities:

    1. The Sequence Landscape of genetic information (>is greater than)

    which generates the

    2. Protein Landscape of amino acids, who fold and form global structures
    (>is also greater than)

    which generates the

    3. Function Landscape of possible biological activities.

    Now, landscape one is greater encompassing in relationship, by nature, than
    either Landscape 2 or 3 when you require the outcome to be relevant
    biologically active function that confers selective advantage, which is all
    that we are interested in terms of the evolution or (not possible)-evolution
    of biological information. Function landscapes are narrow in comparison to
    sequence landscapes, not all sequences can lead to biologically active
    functions. (For example, some percentage of landscape one forms insoluble
    proteins which cannot perform any function.) This also requires an
    environment consideration: protein sequences that could not perform a role
    at normal physiological conditions may be super-active at
    non-physiologically relevant conditions such as extreme salt, heat, pH, etc.
    (for halobacter, extremes will be different than mammalian tissue culture
    but you get the idea.) The question is, out of the sequence landscape, what
    percentage of sequences are there which generate proteins that form folded,
    global structures and that ultimately lead to biologically active functions
    within the function landscape and confer selective advantage. The
    requirements of function and selective advantage, mandated by evolutionary
    developed processes, places strict requirements on the output, that greatly
    narrows (imo) the sequence landscape available for the access of biological
    information. In fact, we currently know that all of life samples only an
    infinitesimally small amount of the available sequence landscape. Whether
    these sequences are only islands of function that stand above the sea of
    modern-day requisite biological function verses the lower threshold function
    of pre-biotic or ancient precursor biotic system function is a
    largely-unaddressed and completely-open-to-interpretation question, imo.

    Josh

    _________________________________________________________________
    STOP MORE SPAM with the new MSN 8 and get 2 months FREE*
    http://join.msn.com/?page=features/junkmail



    This archive was generated by hypermail 2.1.4 : Tue Nov 12 2002 - 14:08:01 EST